NURS 6501 PEDIATRICS MODULE 8

Walden University  NURS 6501 PEDIATRICS MODULE 8  – Step-By-Step Guide

This guide will demonstrate how to complete the Walden University  NURS 6501 PEDIATRICS MODULE 8   assignment based on general principles of academic writing. Here, we will show you the A, B, Cs of completing an academic paper, irrespective of the instructions. After guiding you through what to do, the guide will leave one or two sample essays at the end to highlight the various sections discussed below.

How to Research and Prepare for  NURS 6501 PEDIATRICS MODULE 8  

Whether one passes or fails an academic assignment such as the Walden University  NURS 6501 PEDIATRICS MODULE 8   depends on the preparation done beforehand. The first thing to do once you receive an assignment is to quickly skim through the requirements. Once that is done, start going through the instructions one by one to clearly understand what the instructor wants. The most important thing here is to understand the required format—whether it is APA, MLA, Chicago, etc.

After understanding the requirements of the paper, the next phase is to gather relevant materials. The first place to start the research process is the weekly resources. Go through the resources provided in the instructions to determine which ones fit the assignment. After reviewing the provided resources, use the university library to search for additional resources. After gathering sufficient and necessary resources, you are now ready to start drafting your paper.

How to Write the Introduction for  NURS 6501 PEDIATRICS MODULE 8  

The introduction for the Walden University  NURS 6501 PEDIATRICS MODULE 8   is where you tell the instructor what your paper will encompass. In three to four statements, highlight the important points that will form the basis of your paper. Here, you can include statistics to show the importance of the topic you will be discussing. At the end of the introduction, write a clear purpose statement outlining what exactly will be contained in the paper. This statement will start with “The purpose of this paper…” and then proceed to outline the various sections of the instructions.

How to Write the Body for  NURS 6501 PEDIATRICS MODULE 8  

After the introduction, move into the main part of the  NURS 6501 PEDIATRICS MODULE 8   assignment, which is the body. Given that the paper you will be writing is not experimental, the way you organize the headings and subheadings of your paper is critically important. In some cases, you might have to use more subheadings to properly organize the assignment. The organization will depend on the rubric provided. Carefully examine the rubric, as it will contain all the detailed requirements of the assignment. Sometimes, the rubric will have information that the normal instructions lack.

Another important factor to consider at this point is how to do citations. In-text citations are fundamental as they support the arguments and points you make in the paper. At this point, the resources gathered at the beginning will come in handy. Integrating the ideas of the authors with your own will ensure that you produce a comprehensive paper. Also, follow the given citation format. In most cases, APA 7 is the preferred format for nursing assignments.

How to Write the Conclusion for  NURS 6501 PEDIATRICS MODULE 8  

After completing the main sections, write the conclusion of your paper. The conclusion is a summary of the main points you made in your paper. However, you need to rewrite the points and not simply copy and paste them. By restating the points from each subheading, you will provide a nuanced overview of the assignment to the reader.

How to Format the References List for  NURS 6501 PEDIATRICS MODULE 8  

The very last part of your paper involves listing the sources used in your paper. These sources should be listed in alphabetical order and double-spaced. Additionally, use a hanging indent for each source that appears in this list. Lastly, only the sources cited within the body of the paper should appear here.

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Sample Answer for NURS 6501 PEDIATRICS MODULE 8

Acute Lymphoblastic Leukemia (ALL), which is the case for NURS 6501 PEDIATRICS MODULE 8, is a type of cancer that primarily affects the blood and bone marrow. It is characterized by the uncontrolled proliferation of immature white blood cells, specifically lymphoblasts, which are a type of white blood cell involved in the immune system. In a healthy individual, these cells mature into different types of lymphocytes that play crucial roles in defending the body against infections. 

In ALL, the abnormal lymphoblasts accumulate in the bone marrow, interfering with the production of normal blood cells, including red blood cells, white blood cells, and platelets. As a result, the bone marrow becomes overcrowded with cancerous cells, leading to a decrease in the production of healthy blood cells. 

The symptoms described in the scenario, such as fatigue, paleness, bruising, bone pain, and fever, are common manifestations of ALL. The reduced number of red blood cells (anemia) leads to fatigue and pallor, while the decreased platelet count contributes to easy bruising and bleeding. The bone pain could be a result of the overcrowding of the bone marrow with cancerous cells. 

Sample Answer 2 for NURS 6501 PEDIATRICS MODULE 8

Acute Renal Failure (ARF) can occur in some patients with Acute Lymphoblastic Leukemia (ALL) due to multiple factors. The rapid breakdown of leukemic cells, often seen in the initial stages of treatment, can result in Tumor Lysis Syndrome (TLS), leading to an overload of electrolytes and waste products in the bloodstream that overwhelms the kidneys’ filtering capacity. Additionally, chemotherapy, while targeting cancer cells, can inadvertently damage healthy cells including those in the kidneys, contributing to kidney dysfunction. Leukemic cells infiltrating the kidneys can disrupt their normal function, infections and sepsis can trigger a systemic inflammatory response affecting kidney blood flow, and the overall physical stress of ALL symptoms, such as reduced fluid intake and vomiting, can lead to dehydration and hypovolemia. This combination of factors can lead to renal failure, characterized by elevated blood urea nitrogen (BUN) and creatinine levels, requiring prompt and targeted intervention to manage kidney dysfunction alongside the treatment of ALL. 

Sample Answer 3 for NURS 6501 PEDIATRICS MODULE 8

Acute Sickle Cell Crisis in Sickle Cell Disease (SCD) is characterized by the blockage of small blood vessels due to clumping of misshapen red blood cells, causing reduced blood flow and tissue oxygenation. This vaso-occlusion triggers inflammation, activating pain receptors in the affected areas, resulting in severe pain. Additionally, the cycle of blockage and release during vaso-occlusion leads to tissue damage and further inflammation, intensifying the pain. This complex process involving vaso-occlusion, inflammation, tissue ischemia, and pain receptor activation collectively makes pain the predominant feature of acute SCD crises. 

Sample Answer 4 for NURS 6501 PEDIATRICS MODULE 8

Sickle Cell Disease (SCD) is caused by a genetic mutation in the beta-globin gene, resulting in the production of abnormal hemoglobin S (HbS). HbS causes red blood cells to become rigid and take on a sickle shape, leading to blockages in blood vessels (vaso-occlusion), reduced oxygen delivery, tissue damage, and acute pain episodes. SCD is inherited in an autosomal recessive manner, requiring two copies of the mutated gene for the disease to manifest. Carriers of one normal and one mutated gene have sickle cell trait. 

Sample Answer 5 for NURS 6501 PEDIATRICS MODULE 8

  Hemophilia is a genetic bleeding disorder where there is a deficiency or dysfunction of a specific clotting factor (factor VIII in hemophilia A or factor IX in hemophilia B). This disruption in the clotting cascade leads to ineffective formation of stable blood clots. As a result, individuals with hemophilia experience prolonged bleeding after injuries or trauma, as well as spontaneous bleeding into joints and soft tissues. Diagnosis involves measuring clotting factor levels, and treatment includes replacing the deficient clotting factor to control bleeding and prevent complications.

NURS 6501 Week 8 Case Study Analysis A

Introduction 

The case concerns a 64-year-old Caucasian woman who saw a medical professional because of ongoing back discomfort for the last three weeks. The patient, who was having a pain intensity of 5/10 and occasional acute discomfort, had previously tried self-management with over-the-counter analgesics but did not get considerable alleviation. An extensive analysis uncovered a compression fracture of the second lumbar vertebra (L2), providing insight into a possible musculoskeletal disorder. This case involves a patient with a history of osteoporosis and prescription drugs for bone health. It stimulates an investigation of the complex relationship between musculoskeletal pathophysiology, racial/ethnic factors, and how these together affect the patient’s clinical presentation. Osteoporosis, a disorder marked by reduced bone density, plays a crucial role in comprehending the cause of vertebral fractures and their impact on the patient’s health. Furthermore, recognizing possible racial/ethnic characteristics guarantees a thorough approach to healthcare, considering personalized risk factors and contributing factors. This case study aims to analyze the intricacies of the patient’s illness, offering insights into the underlying pathophysiological processes and their wider implications for therapeutic treatment. 

Musculoskeletal Pathophysiologic Processes: 

The discomfort that the patient is feeling in their back, particularly in connection with a recent compression fracture of the L2 vertebrae, is certainly connected with osteoporosis. This is especially true in light of the fact that bone density has decreased. Osteoporosis is a medical disorder that is marked by a loss in bone density and quality, which increases the vulnerability of bones to fractures. This condition is characterized by a decrease in bone density. In this specific instance, the weakening spine led to the development of a case of compression fracture (Puntillo et al., 2021). Orthoporosis is a degenerative condition that causes changes in the alignment of the spine and an increased risk of fractures. Anterior wedging of the thoracic vertebrae may be an indication of the progressive nature of osteoporosis. 

Racial/Ethnic Variables: 

It is well recognised that people of all racial and ethnic origins are susceptible to developing osteoporosis; nevertheless, the incidence of this condition varies from person to person. The risk of osteoporosis and fractures is greater for Caucasian women in particular, according to the findings of a recent study. It is possible that this is due to a combination of causes, including decreased bone density and hormonal shifts that occur after menopause (Drevdahl et al., 2001). Nevertheless, it is of the utmost importance to acknowledge that osteoporosis may affect people of a wide range of racial and ethnic origins. It is also important to take into consideration the unique risk factors, lifestyle, and genetic variables that are associated with the patient (Drevdahl et al., 2001). 

Interaction of Processes: 

In order to have a complete grasp of the patient’s symptoms, it is essential to comprehend the connection between osteoporosis and the spinal compression fracture. When osteoporosis is present, the bone structure becomes more fragile, which increases the risk of fractures, particularly in places that bear weight. It is believed that the patient’s back discomfort and the reported decrease in her capacity to do activities of daily living (ADLs) are both caused by the compression fracture that occurred in the thoracic spine. The severe pain that the patient is experiencing may also be the result of the fracture, which may create local inflammation and irritation (Imamudeen et al., 2022). In addition, the decreased vertebral integrity may have an effect on the alignment of the spine, which may have an adverse effect on motor function and make the pain even worse (Imamudeen et al., 2022). 

Conclusion 

When it comes to the process of creating symptoms that are associated with the musculoskeletal system, the example highlights the connectivity between osteoporosis and vertebral fractures. It is vital to have an awareness of the pathophysiological processes that are underlying the illness in order to offer full therapy and care for the patient. Additionally, it is important to be aware of the various racial and ethnic variances that may be present. 

References 

Puntillo, F., Giglio, M., Paladini, A., Perchiazzi, G., Viswanath, O., Urits, I., Sabbà, C., Varrassi, G., & Brienza, N. (2021, January). Pathophysiology of musculoskeletal pain: a narrative review. Therapeutic Advances in Musculoskeletal Disease, 13, 1759720X2199506. https://doi.org/10.1177/1759720×21995067 

Drevdahl, D., Taylor, J. Y., & Phillips, D. A. (2001, September). Race and Ethnicity as Variables in Nursing Research, 1952???2000. Nursing Research, 50(5), 305-313 https://doi.org/10.1097/00006199-200109000-00009

Imamudeen, N., Basheer, A., Iqbal, A. M., Manjila, N., Haroon, N. N., & Manjila, S. (2022, April 27). Management of Osteoporosis and Spinal Fractures: Contemporary Guidelines and Evolving Paradigms. Clinical Medicine &   Research, 20(2), 95–106. https://doi.org/10.3121/cmr.2021.1612